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PMID: 22235286 Published · ppublish English Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S.

Intra- and inter-tumor heterogeneity of BRAF(V600E))mutations in primary and metastatic melanoma.

PloS one ·Vol. 7 ·No. 1 ·2012-00-00 ·Pages e29336

Yancovitz M, Litterman A, Yoon J, Ng E, Shapiro RL, Berman RS, Pavlick AC, Darvishian F, Christos P, Mazumdar M, Osman I, Polsky D

Abstract

The rationale for using small molecule inhibitors of oncogenic proteins as cancer therapies depends, at least in part, on the assumption that metastatic tumors are primarily clonal with respect to mutant oncogene. With the emergence of BRAF(V600E) as a therapeutic target, we investigated intra- and inter-tumor heterogeneity in melanoma using detection of the BRAF(V600E) mutation as a marker of clonality. BRAF mutant-specific PCR (MS-PCR) and conventional sequencing were performed on 112 tumors from 73 patients, including patients with matched primary and metastatic specimens (n = 18). Nineteen patients had tissues available from multiple metastatic sites. Mutations were detected in 36/112 (32%) melanomas using conventional sequencing, and 85/112 (76%) using MS-PCR. The better sensitivity of the MS-PCR to detect the mutant BRAF(V600E) allele was not due to the presence of contaminating normal tissue, suggesting that the tumor was comprised of subclones of differing BRAF genotypes. To determine if tumor subclones were present in individual primary melanomas, we performed laser microdissection and mutation detection via sequencing and BRAF(V600E)-specific SNaPshot analysis in 9 cases. Six of these cases demonstrated differing proportions of BRAF(V600E)and BRAF(wild-type) cells in distinct microdissected regions within individual tumors. Additional analyses of multiple metastatic samples from individual patients using the highly sensitive MS-PCR without microdissection revealed that 5/19 (26%) patients had metastases that were discordant for the BRAF(V600E) mutation. In conclusion, we used highly sensitive BRAF mutation detection methods and observed substantial evidence for heterogeneity of the BRAF(V600E) mutation within individual melanoma tumor specimens, and among multiple specimens from individual patients. Given the varied clinical responses of patients to BRAF inhibitor therapy, these data suggest that additional studies to determine possible associations between clinical outcomes and intra- and inter-tumor heterogeneity could prove fruitful.

MeSH Terms
Base Sequence DNA Mutational Analysis Humans Melanoma/genetics,pathology,secondary Microdissection Mutation Neoplasm Metastasis Polymerase Chain Reaction Proto-Oncogene Proteins B-raf/genetics
Chemicals
BRAF protein, human Proto-Oncogene Proteins B-raf
Authors & Affiliations
12 authors, click to expand affiliations / ORCID
Yancovitz Molly
Department of Dermatology, New York University Langone Medical Center, New York, New York, United States of America.
Litterman Adam
Yoon Joanne
Ng Elise
Shapiro Richard L
Berman Russell S
Pavlick Anna C
Darvishian Farbod
Christos Paul
Mazumdar Madhu
Osman Iman
Polsky David
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Article Info
Journal
PloS one
Abbr.
PLoS One
ISSN
1932-6203
Published
2012-00-00
Epub
2012-00-03
Pages
e29336
Language
English
Region
United States
NLM ID
101285081
PMCID
PMC3250426
Subset
IM
Grants
NCI NIH HHS · P30 CA016087 · United States
NCI NIH HHS · 5P30CA16087-31 · United States
NIAMS NIH HHS · T32 AR007190 · United States
NCI NIH HHS · R21 CA109388 · United States
NIAMS NIH HHS · T32 AR07190 - 31 · United States
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