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PMID: 19001857 Published · ppublish English Journal Article Research Support, N.I.H., Extramural Research Support, U.S. Gov't, Non-P.H.S.

mTOR regulates autophagy-associated genes downstream of p73.

Autophagy ·Vol. 5 ·No. 1 ·2009-01-00 ·Pages 114-6

Rosenbluth JM, Pietenpol JA

Abstract

The p53 family consists of three transcription factors, p53, p63 and p73 that share domain architecture and sequence identity. The mTOR (mammalian target of rapamycin) kinase responds to growth factors and nutrient levels to regulate cellular growth and autophagy. Whereas p53 acts both upstream and downstream of mTOR, gene signature-based analyses have revealed that p73 is inhibited by mTOR activity. p53 can both activate and repress autophagy levels depending on cellular context. While less is known about p73, recent studies have shown that it induces cellular autophagy and multiple autophagy-associated genes downstream of mTOR. Chromatin immunoprecipitation analyses demonstrate that endogenous p73 binds the regulatory regions of genes such as ATG5, ATG7 and UVRAG. How p73 regulates the expression levels of these genes in response to different cellular stresses remains unknown. Because p53 family members play key roles in tumor suppression, development, aging and neurodegeneration, the context and manner by which these transcription factors regulate autophagy may have implications for a wide range of human diseases.

MeSH Terms
Animals Autophagy/genetics Base Sequence Gene Expression Regulation Humans Mice Molecular Sequence Data Protein Kinases/metabolism TOR Serine-Threonine Kinases Tumor Suppressor Proteins/metabolism
Chemicals
Tumor Suppressor Proteins Protein Kinases MTOR protein, human mTOR protein, mouse TOR Serine-Threonine Kinases
Authors & Affiliations
2 authors, click to expand affiliations / ORCID
Rosenbluth Jennifer M
Department of Biochemistry, Center in Molecular Toxicology, Vanderbilt-Ingram Cancer Center, Vanderbilt University School of Medicine, Nashville, TN 37232, USA.
Pietenpol Jennifer A
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Article Info
Journal
Autophagy
Abbr.
Autophagy
ISSN
1554-8635
Published
2009-01-00
Epub
2009-00-30
Pages
114-6
Language
English
Region
United States
NLM ID
101265188
PMCID
PMC2792737
Subset
IM
Grants
NIGMS NIH HHS · GM07347 · United States
NIEHS NIH HHS · P30 ES000267-389011 · United States
NCI NIH HHS · R01 CA105436 · United States
NCI NIH HHS · R01 CA070856 · United States
NIGMS NIH HHS · T32 GM007347 · United States
NCI NIH HHS · R01 CA070856-13 · United States
NCI NIH HHS · R01 CA105436-05 · United States
NIEHS NIH HHS · ES00267 · United States
NIEHS NIH HHS · P30 ES000267 · United States
NCI NIH HHS · CA105436 · United States
NCI NIH HHS · P30 CA068485 · United States
NCI NIH HHS · P30 CA068485-13 · United States
NCI NIH HHS · CA68485 · United States
NCI NIH HHS · CA70856 · United States
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