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PMID: 16185251 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't Review

The interleukin-1 signalling pathway in astrocytes: a key contributor to inflammation in the brain.

Journal of anatomy ·Vol. 207 ·No. 3 ·2005-09-00 ·Pages 265-9

Moynagh PN

Abstract

A dysregulated inflammatory response in the central nervous system (CNS) lies at the heart of many neuropathological conditions such as multiple sclerosis and Alzheimer's disease. A key component of these inflammatory conditions is the accumulation of leukocytes in the CNS. The infiltration of leukocytes into the brain is dependent on the induction of leukocyte adhesion molecules and chemoattractant chemokines. Recent studies have suggested the astrocyte to be a key cell in mediating the inflammatory process in the brain and in expressing adhesion molecules and chemokines. Here I overview work in my laboratory and others that demonstrates interleukin-1 (IL-1) to be a key inducer of the expression of these molecules in astrocytes. The temporal expression is sustained in nature and this is due to prolonged activation of the transcription factor NFkappaB. The molecular basis to the sustained activation of NFkappaB is also discussed. The IL-1 signalling pathway thus emerges as a valuable therapeutic target in the treatment of presently incurable neuropathological conditions.

MeSH Terms
Astrocytes/immunology Brain/immunology Cell Adhesion Molecules/metabolism Chemotaxis, Leukocyte Encephalitis/immunology Humans Interleukin-1/antagonists & inhibitors,genetics,metabolism NF-kappa B/metabolism Neurodegenerative Diseases/genetics,immunology,therapy Polymorphism, Genetic Signal Transduction/physiology
Chemicals
Cell Adhesion Molecules Interleukin-1 NF-kappa B
Authors & Affiliations
1 authors, click to expand affiliations / ORCID
Moynagh Paul N
Department of Pharmacology, Conway Institute of Biomolecular and Biomedical Research, University College Dublin, Ireland. P.Moynagh@ucd.i.e
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Article Info
Journal
Journal of anatomy
Abbr.
J Anat
ISSN
0021-8782
Published
2005-09-00
Pages
265-9
Language
English
Region
England
NLM ID
0137162
PMCID
PMC1571539
Subset
IM
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