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PMID: 9409474 Published · ppublish English Journal Article Research Support, U.S. Gov't, P.H.S.

Reduced outward K+ conductances generate depolarizing after-potentials in rat supraoptic nucleus neurones.

The Journal of physiology ·Vol. 505 ( Pt 1) ·1997-11-15 ·Pages 95-106

Li Z, Hatton GI

Abstract

1. Whole-cell patch clamp recordings were obtained from sixty-five rat supraoptic nucleus (SON) neurones in brain slices to investigate ionic mechanisms underlying depolarizing after-potentials (DAPs). When cells were voltage clamped around -58 mV, slow inward currents mediating DAPs (IDAP), evoked by three brief depolarizing pulses, had a peak of 17 +/- 1 pA (mean +/- S.E.M.) and lasted for 2.8 +/- 0.1 s. 2. No significant differences in the amplitude and duration were observed when one to three preceding depolarizing pulses were applied, although there was a tendency for twin pulses to evoke larger IDAP than a single pulse. The IDAP was absent when membrane potentials were more negative than -70 mV. In the range -70 to -50 mV, IDAP amplitudes and durations increased as the membrane became more depolarized, with an activation threshold of -65.7 +/- 0.7 mV. 3. IDAP with normal amplitude and duration could be evoked during the decay of a preceding IDAP. As frequencies of depolarizing pulses rose from 2 to 20 Hz, the times to peak IDAP amplitude were reduced but the amplitudes and durations did not change. 4. A consistent reduction in membrane conductance during the IDAP was observed in all SON neurones tested, and averaged 34.6 +/- 3.3%. Small hyperpolarizing pulses used to measure membrane conductances appeared not to disturb major ionic mechanisms underlying IDAP, since the slope and duration of IDAP with and without test pulses were similar. 5. The IDAP had an averaged reversal potential of -87.4 +/- 1.6 mV, which was close to the K+ equilibrium potential. An elevation in [K+]o reduced or abolished the IDAP, and shifted its reversal potential toward more positive levels. Perifusion of slices with 7.5-10 mM TEA, a K+ channel blocker, reversibly suppressed the IDAP. 6. Both Na+ and Ca2+ currents failed to induce an IDAP-like current during perifusion of slices with media containing high [K+]o or TEA. However, the IDAP was abolished by replacing external Ca2+ with Co2+, or replacing 82% of external Na+ with choline or Li+. Perifusion of slices with media containing 1-2 microM TTX also reduced IDAP by 55.5 +/- 9.0%. 7. These results suggest that the generation of DAPs in SON neurones mainly involves a reduction in outward K+ current(s), which probably has little or no inactivation and can be inhibited by [Ca2+]i transients, due to Ca2+ influx during action potentials and Ca2+ release from internal stores. Na+ influx might provide a permissive influence for Ca(2+)-induced reduction of K+ conductances and/or help to raise [Ca2+]i via reverse-mode Ca(2+)-Na+ exchange. Other conductances, making minor contributions to the IDAP, may also be involved.

MeSH Terms
Animals Calcium/metabolism,pharmacology Electrophysiology Male Membrane Potentials/physiology Neurons/physiology Patch-Clamp Techniques Potassium/metabolism Potassium Channels/physiology Rats Rats, Sprague-Dawley Sodium/metabolism Supraoptic Nucleus/physiology Tetraethylammonium/pharmacology Tetrodotoxin/pharmacology
Chemicals
Potassium Channels Tetrodotoxin Tetraethylammonium Sodium Potassium Calcium
Authors & Affiliations
2 authors, click to expand affiliations / ORCID
Li Z
Department of Neuroscience, University of California at Riverside 92521, USA.
Hatton G I
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Article Info
Journal
The Journal of physiology
Abbr.
J Physiol
ISSN
0022-3751
Published
1997-11-15
Pages
95-106
Language
English
Region
England
NLM ID
0266262
PMCID
PMC1160096
Subset
IM
Grants
NINDS NIH HHS · NS09140 · United States
NINDS NIH HHS · NS16942 · United States
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