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PMID: 9116295 Published · ppublish English Journal Article

Frequent hypermethylation of p16 and p15 genes in multiple myeloma.

Blood ·Vol. 89 ·No. 7 ·1997-04-01 ·Pages 2500-6

Ng MH, Chung YF, Lo KW, Wickham NW, Lee JC, Huang DP

Abstract

Both p16 and p15, encoded by the genes located on chromosome 9p21, are inhibitors of cyclin-dependent kinases (CDK4/6) and the upstream regulators of Rb function. In hematopoietic malignancies, deletion of p16/p15 locus has been shown to be highly specific to lymphoid, and more particularly from B-lineage malignancies except multiple myeloma (MM). To investigate whether these genes are inactivated by deletions, mutations, and hypermethylation of the 5' CpG islands, we examined 12 MM patients by Southern hybridization and polymerase chain reaction-single-strand conformation polymorphism (PCR-SSCP) analysis. No deletions nor mutations of the p16 and p15 genes were found. However, hypermethylation was observed in 75% for p16 and 67% for p15 in our group of MM patients. Such high frequencies of involvement of these genes in MM make them hitherto the most common genetic abnormalities in this disease. Concomitant hypermethylation, uncommon thus far in the literature of the study of these genes, is a rather common phenomenon, occurring in 67% of our patient group. Moreover, hypermethylation of p16/p15 was associated with blastic disease and concomitant hypermethylation of both genes may be pathogenetically related to plasmacytoma development. These results indicate that these genes are important in MM pathogenesis. Here we report, for the first time in the literature, the high incidences of p16 and p15 alterations in MM, not by homozygous deletions or mutations, but solely by hypermethylation of the 5' CpG islands, which may be a specific mechanism in this disease.

MeSH Terms
Adult Aged Aged, 80 and over Biomarkers, Tumor Blotting, Southern Carrier Proteins/genetics Cell Cycle/genetics Cell Cycle Proteins Cell Transformation, Neoplastic CpG Islands Cyclin-Dependent Kinase Inhibitor p15 Cyclin-Dependent Kinase Inhibitor p16 DNA Methylation DNA, Neoplasm/chemistry,genetics Female Humans Male Middle Aged Multiple Myeloma/genetics,pathology Polymerase Chain Reaction Polymorphism, Single-Stranded Conformational Tumor Suppressor Proteins
Chemicals
Biomarkers, Tumor CDKN2B protein, human Carrier Proteins Cell Cycle Proteins Cyclin-Dependent Kinase Inhibitor p15 Cyclin-Dependent Kinase Inhibitor p16 DNA, Neoplasm Tumor Suppressor Proteins
Authors & Affiliations
6 authors, click to expand affiliations / ORCID
Ng M H
Department of Anatomical & Cellular Pathology, The Chinese University of Hong Kong, Shatin.
Chung Y F
Lo K W
Wickham N W
Lee J C
Huang D P
Article Info
Journal
Blood
Abbr.
Blood
ISSN
0006-4971
Published
1997-04-01
Pages
2500-6
Language
English
Region
United States
NLM ID
7603509
Subset
IM
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