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PMID: 9059332 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, P.H.S. Review

Reciprocal paracrine interactions between tumour cells and endothelial cells: the 'angiogenesis progression' hypothesis.

European journal of cancer (Oxford, England : 1990) ·Vol. 32A ·No. 14 ·1996-12-00 ·Pages 2438-50

Rak J, Filmus J, Kerbel RS

Abstract

暂无摘要

MeSH Terms

Animals Colorectal Neoplasms/blood supply Disease Progression Endothelial Growth Factors/metabolism Endothelium, Vascular/physiopathology Genes, ras/physiology Humans Lymphokines/metabolism Melanoma/blood supply Mice Neoplasm Metastasis Neoplasms/blood supply Neovascularization, Pathologic/physiopathology Rats Skin Neoplasms/blood supply Vascular Endothelial Growth Factor A Vascular Endothelial Growth Factors

Chemicals

Endothelial Growth Factors Lymphokines Vascular Endothelial Growth Factor A Vascular Endothelial Growth Factors

Authors & Affiliations

3 authors, click to expand affiliations / ORCID

Rak J

Cancer Biology Research Division, Sunnybrook Health Science Center, Toronto, Ontario, Canada.

Filmus J

Kerbel R S

Article Info

Journal

European journal of cancer (Oxford, England : 1990)

Abbr.

Eur J Cancer

ISSN

0959-8049

DOI

10.1016/s0959-8049(96)00396-6

Published

1996-12-00

Pages

2438-50

Language

English

Region

England

NLM ID

9005373

Subset

Grants

NCI NIH HHS · CA 4233 · United States

External Links

PubMed source DOI: 10.1016/s0959-8049(96)00396-6 View journal details

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Reciprocal paracrine interactions between tumour cells and endothelial cells: the 'angiogenesis progression' hypothesis.

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