Metabolism of third complement component (C3) in nephritis. Involvement of the classic and alternate (properdin) pathways for complement activation.

The New England journal of medicine ·Vol. 287 ·No. 17 ·1972-10-26 ·Pages 835-40

Hunsicker LG, Ruddy S, Carpenter CB, Schur PH, Merrill JP, Müller-Eberhard HJ, Austen KF

Abstract

暂无摘要

MeSH Terms

Adult Chronic Disease Complement System Proteins/analysis,metabolism Female Glomerulonephritis/immunology,metabolism Humans Immunodiffusion Immunoelectrophoresis Iodine Isotopes Lupus Erythematosus, Systemic/immunology,metabolism Male Middle Aged Nephritis/immunology,metabolism Properdin/analysis,metabolism Protein Binding

Chemicals

Iodine Isotopes Properdin Complement System Proteins

Authors & Affiliations

7 authors, click to expand affiliations / ORCID

Hunsicker L G

Ruddy S

Carpenter C B

Schur P H

Merrill J P

Müller-Eberhard H J

Austen K F

Article Info

Journal

The New England journal of medicine

Abbr.

N Engl J Med

ISSN

0028-4793

DOI

10.1056/NEJM197210262871701

Published

1972-10-26

Pages

835-40

Language

English

Region

United States

NLM ID

0255562

Subset

External Links

PubMed source DOI: 10.1056/NEJM197210262871701 View journal details

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Metabolism of third complement component (C3) in nephritis. Involvement of the classic and alternate (properdin) pathways for complement activation.

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