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PMID: 3260256 Published · ppublish English Journal Article Research Support, U.S. Gov't, P.H.S.

IL-1 inhibits B cell differentiation in long term bone marrow cultures.

Journal of immunology (Baltimore, Md. : 1950) ·Vol. 141 ·No. 2 ·1988-07-15 ·Pages 531-8

Dorshkind K

Abstract

There is evidence that stromal cells are responsive to changes in their external milieu and that this can affect their function. IL-1 has been identified as one mediator that can affect stromal cells by increasing their secretion of CSF. The monokine has also been reported to be a B cell differentiation factor. The purpose of this study was to test the effects of IL-1 on the pattern of hemopoietic cell differentiation by adding IL-1 alpha to myeloid long term bone marrow cultures (MBMC) at the time of their transfer to lymphoid bone marrow culture conditions. This usually results in the cessation of myelopoiesis and the induction of B lymphopoiesis. The addition of 50 U/ml of rIL-1 alpha, but not 10 U/ml, to MBMC at the time of their transfer to lymphoid conditions resulted in a complete inhibition of B cell differentiation and sustained myelopoiesis. To determine whether adherent layer cells contributed to this effect, conditioned medium (CM) was collected from adherent layers treated previously with the antibiotic mycophenolic acid. This depletes the hemopoietic cells from the cultures and retains a purified population of stromal cells. CM from mycophenolic acid- treated adherent layers exposed for 24 h to 50 U/ml of IL-1 was added at volume concentrations of 5, 10, and 25% to MBMC at the time of transfer to lymphoid bone marrow culture conditions and at each feeding thereafter. Expression of the B lineage associated 14.8 Ag and IgM was inhibited on a dose dependent basis, and myelopoiesis was sustained in cultures to which 25% CM had been added. Induction of B lymphopoiesis occurred in cultures to which adherent cell CM not exposed to IL-1 had been added. The CM from the IL-1-treated adherent cells contained CSF, because it promoted the growth of myeloid colonies from fresh marrow or MBMC cells and stimulated the granulocyte-macrophage-CSF sensitive FDC-P1 cell line to proliferate. IL-3 was not present in the CM, because stimulation of the IL-3 sensitive 32D cell line was not observed. The CM from the IL-1-treated adherent cells stimulated thymocytes to proliferate in the presence of PHA. This raised the possibility that the induced CSF may have required IL-1 to mediate their effects in the cultures. However, B lymphopoiesis was inhibited and myelopoiesis maintained upon addition of recombinant granulocyte-, macrophage-, and granulocyte-macrophage-CSF to cultures, indicating that IL-1 or other non-CSF molecules induced by it need not be present.(ABSTRACT TRUNCATED AT 400 WORDS)

MeSH Terms
Animals B-Lymphocytes/cytology,drug effects Bone Marrow/metabolism Bone Marrow Cells Cell Adhesion Cells, Cultured Colony-Stimulating Factors/metabolism Culture Media Female Growth Inhibitors/pharmacology Hematopoiesis/drug effects Interleukin-1/pharmacology Lymphocyte Activation Male Mice Mice, Inbred BALB C Recombinant Proteins/pharmacology T-Lymphocytes/immunology Time Factors
Chemicals
Colony-Stimulating Factors Culture Media Growth Inhibitors Interleukin-1 Recombinant Proteins
Authors & Affiliations
1 authors, click to expand affiliations / ORCID
Dorshkind K
Division of Biomedical Sciences, University of California, Riverside 92521.
Article Info
Journal
Journal of immunology (Baltimore, Md. : 1950)
Abbr.
J Immunol
ISSN
0022-1767
Published
1988-07-15
Pages
531-8
Language
English
Region
United States
NLM ID
2985117R
Subset
IM
Grants
NIAID NIH HHS · AI00843 · United States
NIAID NIH HHS · AI21256 · United States
NHLBI NIH HHS · HL36591 · United States
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