Home LiteratureArticle Details

PMID: 2497346 Published · ppublish English Journal Article Research Support, U.S. Gov't, P.H.S.

A defect in mitochondrial electron-transport activity (NADH-coenzyme Q oxidoreductase) in Leber's hereditary optic neuropathy.

The New England journal of medicine ·Vol. 320 ·No. 20 ·1989-05-18 ·Pages 1331-3

Parker WD, Oley CA, Parks JK

Abstract

暂无摘要

MeSH Terms

Electron Transport Hereditary Sensory and Motor Neuropathy/enzymology Humans Male Mitochondria/enzymology,metabolism NAD(P)H Dehydrogenase (Quinone) Optic Atrophies, Hereditary/enzymology,genetics Quinone Reductases/deficiency,genetics Rotenone/metabolism

Chemicals

Rotenone NAD(P)H Dehydrogenase (Quinone) Quinone Reductases

Authors & Affiliations

3 authors, click to expand affiliations / ORCID

Parker W D

Department of Neurology, University of Colorado School of Medicine, Denver.

Oley C A

Parks J K

Article Info

Journal

The New England journal of medicine

Abbr.

N Engl J Med

ISSN

0028-4793

DOI

10.1056/NEJM198905183202007

Published

1989-05-18

Pages

1331-3

Language

English

Region

United States

NLM ID

0255562

Subset

Grants

NINDS NIH HHS · NS25382 · United States

NCRR NIH HHS · RR-69 · United States

External Links

PubMed source DOI: 10.1056/NEJM198905183202007 View journal details

Analysis Services

A defect in mitochondrial electron-transport activity (NADH-coenzyme Q oxidoreductase) in Leber's hereditary optic neuropathy.

Contact

WeChat Official Account