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PMID: 2241624 Published · ppublish English Journal Article Research Support, U.S. Gov't, Non-P.H.S. Research Support, U.S. Gov't, P.H.S. Review

Background review and current concepts of reperfusion injury.

Archives of neurology ·Vol. 47 ·No. 11 ·1990-11-00 ·Pages 1245-54

Hallenbeck JM, Dutka AJ

Abstract

We define the concept of reperfusion injury, and we present a background chronology of experimental work supporting and questioning this concept. We identify several new influences, such as current clinical interest in thrombolytic therapy for acute ischemia of heart and brain and the growing recognition of endothelium as a regulator of homeostasis. We propose that these influences will encourage a reexamination of reperfusion injury as a factor in the ultimate outcome of tissue exposed to reversible ischemia. We briefly discuss the major mechanisms presently implicated in reperfusion injury--loss of calcium homeostasis, free radical generation, leukocyte-mediated injury, and acute hypercholesterolemia.

MeSH Terms
Calcium/metabolism Cholesterol/metabolism Free Radicals Humans Leukocytes/metabolism Oxygen/metabolism Reperfusion Injury/metabolism,physiopathology
Chemicals
Free Radicals Cholesterol Oxygen Calcium
Authors & Affiliations
2 authors, click to expand affiliations / ORCID
Hallenbeck J M
Neurology Department, National Naval Medical Center, Bethesda, MD 20814-5011.
Dutka A J
Article Info
Journal
Archives of neurology
Abbr.
Arch Neurol
ISSN
0003-9942
Published
1990-11-00
Pages
1245-54
Language
English
Region
United States
NLM ID
0372436
Subset
IM
Grants
NINDS NIH HHS · 1 R01 NS28225-01 · United States
Corrections
ErratumIn
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