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PMID: 21705420 Published · ppublish English Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Distinctive genetic and clinical features of CMT4J: a severe neuropathy caused by mutations in the PI(3,5)P₂ phosphatase FIG4.

Brain : a journal of neurology ·Vol. 134 ·No. Pt 7 ·2011-07-00 ·Pages 1959-71

Nicholson G, Lenk GM, Reddel SW, Grant AE, Towne CF, Ferguson CJ, Simpson E, Scheuerle A, Yasick M, Hoffman S, Blouin R, Brandt C, Coppola G, Biesecker LG, Batish SD, Meisler MH

Abstract

Charcot-Marie-Tooth disease is a genetically heterogeneous group of motor and sensory neuropathies associated with mutations in more than 30 genes. Charcot-Marie-Tooth disease type 4J (OMIM 611228) is a recessive, potentially severe form of the disease caused by mutations of the lipid phosphatase FIG4. We provide a more complete view of the features of this disorder by describing 11 previously unreported patients with Charcot-Marie-Tooth disease type 4J. Three patients were identified from a small cohort selected for screening because of their early onset disease and progressive proximal as well as distal weakness. Eight patients were identified by large-scale exon sequencing of an unselected group of 4000 patients with Charcot-Marie-Tooth disease. In addition, 34 new FIG4 variants were detected. Ten of the new CMT4J cases have the compound heterozygous genotype FIG4(I41T/null) described in the original four families, while one has the novel genotype FIG4(L17P/nul)(l). The population frequency of the I41T allele was found to be 0.001 by genotyping 5769 Northern European controls. Thirty four new variants of FIG4 were identified. The severity of Charcot-Marie-Tooth disease type 4J ranges from mild clinical signs to severe disability requiring the use of a wheelchair. Both mild and severe forms have been seen in patients with the same genotype. The results demonstrate that Charcot-Marie-Tooth disease type 4J is characterized by highly variable onset and severity, proximal as well as distal and asymmetric muscle weakness, electromyography demonstrating denervation in proximal and distal muscles, and frequent progression to severe amyotrophy. FIG4 mutations should be considered in Charcot-Marie-Tooth patients with these characteristics, especially if found in combination with sporadic or recessive inheritance, childhood onset and a phase of rapid progression.

MeSH Terms
Adult Australia Charcot-Marie-Tooth Disease/classification,complications,diagnosis,genetics Child Child, Preschool Exons/genetics Family Health Female Flavoproteins/genetics Foot Deformities/etiology,genetics Genotype Glutamic Acid/genetics Humans Lysine/genetics Male Middle Aged Models, Molecular Muscle Weakness/etiology,genetics Mutation/genetics Neural Conduction/genetics Phenotype Phosphoric Monoester Hydrolases Sural Nerve/pathology,ultrastructure
Chemicals
Flavoproteins Glutamic Acid FIG4 protein, human Phosphoric Monoester Hydrolases Lysine
Authors & Affiliations
16 authors, click to expand affiliations / ORCID
Nicholson Garth
Department of Neurology, University of Sydney, ANZAC Institute, Concord Hospital, NSW 2139, Australia.
Lenk Guy M
Reddel Stephen W
Grant Adrienne E
Towne Charles F
Ferguson Cole J
Simpson Ericka
Scheuerle Angela
Yasick Michelle
Hoffman Stuart
Blouin Randall
Brandt Carla
Coppola Giovanni
Biesecker Leslie G
Batish Sat D
Meisler Miriam H
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Article Info
Journal
Brain : a journal of neurology
Abbr.
Brain
ISSN
1460-2156
Published
2011-07-00
Pages
1959-71
Language
English
Region
England
NLM ID
0372537
PMCID
PMC3122378
Subset
IM
Grants
NIMH NIH HHS · R01 MH061675 · United States
NIA NIH HHS · U24 AG021886 · United States
NIMH NIH HHS · R01 MH067257 · United States
NIMH NIH HHS · R01 MH060870 · United States
NIMH NIH HHS · R01 MH059586 · United States
NIMH NIH HHS · R01 MH059566 · United States
NIGMS NIH HHS · R01 GM24872 · United States
NIMH NIH HHS · R01 MH060879 · United States
NIMH NIH HHS · R01 MH059571 · United States
NIMH NIH HHS · R01 MH059565 · United States
NIMH NIH HHS · R01 MH059587 · United States
NIGMS NIH HHS · T32 GM07863 · United States
NIMH NIH HHS · R01 MH063420 · United States
NIA NIH HHS · U24 AG21886 · United States
NIMH NIH HHS · R01 MH059588 · United States
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