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PMID: 2159816 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't

Increased erythroid potentiating activity/tissue inhibitor of metalloproteinases and jun/fos transcription factor complex characterize tumor promoter-induced megakaryoblastic differentiation of K562 leukemia cells.

Blood ·Vol. 75 ·No. 10 ·1990-05-15 ·Pages 1974-82

Alitalo R, Partanen J, Pertovaara L, Hölttä E, Sistonen L, Andersson L, Alitalo K

Abstract

Molecular cloning has revealed that erythroid potentiating activity (EPA) and tissue inhibitor of metalloproteinases (TIMP) represent two distinct activities of a single protein. We have studied the expression of the EPA/TIMP gene at the mRNA and protein levels during 12-O-tetradecanoyl-phorbol-13-acetate (TPA)-induced megakaryoblastic differentiation of K562 human chronic myeloid leukemia cells. Northern hybridization analysis showed that the EPA/TIMP mRNA was increased within 3 hours of TPA-induction and reached maximal levels (about 50-fold induction) during the first day of treatment. The expression of mRNAs for two major metalloproteinases, collagenase-I and stromelysin, were activated in parallel in the differentiation-induced K562 cells. The increase of EPA/TIMP mRNA correlated with increased EPA/TIMP protein biosynthesis and secretion: the TPA-induced cells secreted substantially enhanced amounts of metabolically labeled proteins, of which EPA/TIMP represented up to 50% after the first day of treatment (over 100-fold induction). The induction of EPA/TIMP mRNA was associated with its increased transcription. EPA/TIMP induction required continuous protein synthesis, being completely inhibited by addition of the protein synthesis inhibitor cycloheximide simultaneously with TPA, but only partially inhibited in a time-dependent manner if cycloheximide was added after TPA. Unlike in other cells tested, the jun and c-fos transcription factor mRNAs showed a prolonged biphasic induction response in K562 cells during TPA treatment. This response was associated with enhanced activity of a transfected recombinant reporter plasmid containing binding sites for the jun/fos transcription factor complex (AP-1) similar to the TPA-responsive element (TRE) sequence we found in the EPA/TIMP gene promoter. We suggest that the induction of EPA/TIMP and several other genes specific for the differentiating K562 cells may be a consequence of the sustained activation of immediate early genes encoding transcription factors, such as jun and c-fos.

MeSH Terms
Cell Line Cell Transformation, Neoplastic/drug effects DNA-Binding Proteins/genetics,metabolism Gene Expression/drug effects Glycoproteins/genetics,metabolism Humans Leukemia, Myelogenous, Chronic, BCR-ABL Positive/metabolism,pathology Lymphokines/genetics,metabolism Microbial Collagenase/antagonists & inhibitors Protein Biosynthesis Proto-Oncogene Proteins/genetics,metabolism Proto-Oncogene Proteins c-fos Proto-Oncogene Proteins c-jun RNA, Messenger/genetics Tetradecanoylphorbol Acetate/pharmacology Tissue Inhibitor of Metalloproteinases Transcription Factors/genetics,metabolism
Chemicals
DNA-Binding Proteins Glycoproteins Lymphokines Proto-Oncogene Proteins Proto-Oncogene Proteins c-fos Proto-Oncogene Proteins c-jun RNA, Messenger Tissue Inhibitor of Metalloproteinases Transcription Factors Microbial Collagenase Tetradecanoylphorbol Acetate
Authors & Affiliations
7 authors, click to expand affiliations / ORCID
Alitalo R
Transplantation Laboratory, University of Helsinki, Finland.
Partanen J
Pertovaara L
Hölttä E
Sistonen L
Andersson L
Alitalo K
Article Info
Journal
Blood
Abbr.
Blood
ISSN
0006-4971
Published
1990-05-15
Pages
1974-82
Language
English
Region
United States
NLM ID
7603509
Subset
IM
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