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PMID: 20065189 Published · ppublish English Clinical Trial, Phase II Journal Article Multicenter Study Research Support, Non-U.S. Gov't

Vandetanib for the treatment of patients with locally advanced or metastatic hereditary medullary thyroid cancer.

Wells SA, Gosnell JE, Gagel RF, Moley J, Pfister D, Sosa JA, Skinner M, Krebs A, Vasselli J, Schlumberger M

Abstract

PURPOSE There is no effective therapy for patients with distant metastasis of medullary thyroid carcinoma (MTC). Activating mutations in the RET proto-oncogene cause hereditary MTC, which provides a strong therapeutic rationale for targeting RET kinase activity. This open-label, phase II study assessed the efficacy of vandetanib, a selective oral inhibitor of RET, vascular endothelial growth factor receptor, and epidermal growth factor receptor signaling, in patients with advanced hereditary MTC. METHODS Patients with unresectable locally advanced or metastatic hereditary MTC received initial treatment with once-daily oral vandetanib 300 mg. The dose was adjusted additionally in some patients on the basis of observed toxicity until disease progression or any other withdrawal criterion was met. The primary assessment was objective tumor response (by RECIST [Response Evaluation Criteria in Solid Tumors]). Results Thirty patients received initial treatment with vandetanib 300 mg/d. On the basis of investigator assessments, 20% of patients (ie, six of 30 patients) experienced a confirmed partial response (median duration of response at data cutoff, 10.2 months). An additional 53% of patients (ie, 16 of 30 patients) experienced stable disease at >/= 24 weeks, which yielded a disease control rate of 73% (ie, 22 of 30 patients). In 24 patients, serum calcitonin levels showed a 50% or greater decrease from baseline that was maintained for at least 4 weeks; 16 patients showed a similar reduction in serum carcinoembryonic antigen levels. The most common adverse events were diarrhea (70%), rash (67%), fatigue (63%), and nausea (63%). CONCLUSION In this study, vandetanib demonstrated durable objective partial responses and disease control with a manageable adverse event profile. These results demonstrate that vandetanib may provide an effective therapeutic option in patients with advanced hereditary MTC, a rare disease for which there has been no effective therapy.

MeSH Terms
Administration, Oral Adult Aged Antineoplastic Agents/administration & dosage,adverse effects Biomarkers, Tumor/blood Calcitonin/blood Carcinoembryonic Antigen/blood Carcinoma, Medullary/drug therapy,genetics,metabolism,mortality,secondary Disease-Free Survival Drug Administration Schedule Female France Genetic Predisposition to Disease Germ-Line Mutation Humans Kaplan-Meier Estimate Male Middle Aged Pedigree Piperidines/administration & dosage,adverse effects Protein Kinase Inhibitors/administration & dosage,adverse effects Proto-Oncogene Mas Proto-Oncogene Proteins c-ret/antagonists & inhibitors,genetics Quinazolines/administration & dosage,adverse effects Risk Factors Thyroid Neoplasms/drug therapy,genetics,metabolism,mortality,pathology Time Factors Treatment Outcome United States Young Adult
Chemicals
Antineoplastic Agents Biomarkers, Tumor Carcinoembryonic Antigen MAS1 protein, human Piperidines Protein Kinase Inhibitors Proto-Oncogene Mas Quinazolines Calcitonin Proto-Oncogene Proteins c-ret RET protein, human N-(4-bromo-2-fluorophenyl)-6-methoxy-7-((1-methylpiperidin-4-yl)methoxy)quinazolin-4-amine
Authors & Affiliations
10 authors, click to expand affiliations / ORCID
Wells Samuel A
Dept of Surgery, Washington University School of Medicine, Box 8109, 660 S Euclid Ave, St Louis, MO 63110, USA. wellss@wudosis.wustl.edu
Gosnell Jessica E
Gagel Robert F
Moley Jeffrey
Pfister David
Sosa Julie A
Skinner Michael
Krebs Annetta
Vasselli James
Schlumberger Martin
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Article Info
Journal
Journal of clinical oncology : official journal of the American Society of Clinical Oncology
Abbr.
J Clin Oncol
ISSN
1527-7755
Published
2010-02-10
Epub
2010-00-11
Pages
767-72
Language
English
Region
United States
NLM ID
8309333
PMCID
PMC2834392
Subset
IM
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