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PMID: 19729485 Published · ppublish English Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Endothelial-mesenchymal transition as a novel mechanism for generating myofibroblasts during diabetic nephropathy.

The American journal of pathology ·Vol. 175 ·No. 4 ·2009-10-00 ·Pages 1371-3

Kizu A, Medici D, Kalluri R

Abstract

This Commentary provides perspective on epithelial-mesenchymal transition in diabetic nephropathy.

MeSH Terms
Animals Diabetic Nephropathies/pathology Endothelium/pathology Fibroblasts/pathology Fibrosis Humans Mesoderm/pathology Platelet Endothelial Cell Adhesion Molecule-1/metabolism
Chemicals
Platelet Endothelial Cell Adhesion Molecule-1
Authors & Affiliations
3 authors, click to expand affiliations / ORCID
Kizu Akane
Division of Matrix Biology, Department of Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, MA 02115, USA.
Medici Damian
Kalluri Raghu
References (17)
17 references, click to expand
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Article Info
Journal
The American journal of pathology
Abbr.
Am J Pathol
ISSN
1525-2191
Published
2009-10-00
Epub
2009-00-03
Pages
1371-3
Language
English
Region
United States
NLM ID
0370502
PMCID
PMC2751533
Subset
IM
Grants
NIDDK NIH HHS · DK61688 · United States
NIDDK NIH HHS · DK55001 · United States
NIDDK NIH HHS · R01 DK061688 · United States
NIDDK NIH HHS · R01 DK062987 · United States
NCI NIH HHS · R01 CA125550 · United States
NCI NIH HHS · CA125550 · United States
NIDDK NIH HHS · R01 DK055001 · United States
NIDDK NIH HHS · DK62987 · United States
NIAAA NIH HHS · AA53194 · United States
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