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PMID: 17475894 Published · ppublish English Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

A new population of cells lacking expression of CD27 represents a notable component of the B cell memory compartment in systemic lupus erythematosus.

Journal of immunology (Baltimore, Md. : 1950) ·Vol. 178 ·No. 10 ·2007-05-15 ·Pages 6624-33

Wei C, Anolik J, Cappione A, Zheng B, Pugh-Bernard A, Brooks J, Lee EH, Milner EC, Sanz I

Abstract

Human memory B cells comprise isotype-switched and nonswitched cells with both subsets displaying somatic hypermutation. In addition to somatic hypermutation, CD27 expression has also been considered a universal memory B cell marker. We describe a new population of memory B cells containing isotype-switched (IgG and IgA) and IgM-only cells and lacking expression of CD27 and IgD. These cells are present in peripheral blood and tonsils of healthy subjects and display a degree of hypermutation comparable to CD27+ nonswitched memory cells. As conventional memory cells, they proliferate in response to CpG DNA and fail to extrude rhodamine. In contrast to other recently described CD27-negative (CD27neg) memory B cells, they lack expression of FcRH4 and recirculate in the peripheral blood. Although CD27neg memory cells are relatively scarce in healthy subjects, they are substantially increased in systemic lupus erythematosus (SLE) patients in whom they frequently represent a large fraction of all memory B cells. Yet, their frequency is normal in patients with rheumatoid arthritis or chronic hepatitis C. In SLE, an increased frequency of CD27neg memory cells is significantly associated with higher disease activity index, a history of nephritis, and disease-specific autoantibodies (anti-dsDNA, anti-Smith (Sm), anti-ribonucleoprotein (RNP), and 9G4). These findings enhance our understanding of the B cell diversification pathways and provide mechanistic insight into the immunopathogenesis of SLE.

MeSH Terms
Autoantibodies/biosynthesis,blood B-Lymphocyte Subsets/immunology,metabolism,pathology Biomarkers/blood Cell Proliferation Female Humans Immunoglobulin D/biosynthesis,deficiency,genetics Immunologic Memory/genetics Immunophenotyping Lupus Erythematosus, Systemic/genetics,immunology,pathology Male Membrane Proteins/biosynthesis,deficiency,genetics Palatine Tonsil/immunology,metabolism Severity of Illness Index Tumor Necrosis Factor Receptor Superfamily, Member 7/biosynthesis,deficiency,genetics
Chemicals
Autoantibodies Biomarkers Immunoglobulin D Membrane Proteins Tumor Necrosis Factor Receptor Superfamily, Member 7
Authors & Affiliations
9 authors, click to expand affiliations / ORCID
Wei Chungwen
Department of Medicine, Division of Clinical Immunology and Rheumatology, University of Rochester School of Medicine and Dentistry, NY 14642, USA.
Anolik Jennifer
Cappione Amedeo
Zheng Bo
Pugh-Bernard Aimee
Brooks James
Lee Eun-Hyung
Milner Eric C B
Sanz Iñaki
Article Info
Journal
Journal of immunology (Baltimore, Md. : 1950)
Abbr.
J Immunol
ISSN
0022-1767
Published
2007-05-15
Pages
6624-33
Language
English
Region
United States
NLM ID
2985117R
Subset
IM
Grants
NIAMS NIH HHS · K08AR048303 · United States
NIAID NIH HHS · R01 AI049660-01A1 · United States
NIAID NIH HHS · U19 AI56390 · United States
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