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PMID: 17356379 Published · ppublish English Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

TDP-43-positive white matter pathology in frontotemporal lobar degeneration with ubiquitin-positive inclusions.

Journal of neuropathology and experimental neurology ·Vol. 66 ·No. 3 ·2007-03-00 ·Pages 177-83

Neumann M, Kwong LK, Truax AC, Vanmassenhove B, Kretzschmar HA, Van Deerlin VM, Clark CM, Grossman M, Miller BL, Trojanowski JQ, Lee VM

Abstract

TDP-43 was recently identified as the major disease protein in neuronal inclusions in frontotemporal lobar degeneration with ubiquitin-positive inclusions (FTLD-U). TDP-43 is not only linked to disease mechanisms in FTLD-U, but it is also the most robust marker for the specific detection of neuronal inclusions in FTLD-U. In this study, we describe additional TDP-43 pathology in the white matter as a characteristic feature in a series of 38 FTLD-U cases including 3 cases with mutations in the progranulin gene. White matter pathology was most abundant in frontal and temporal lobes, but it was also detectable in brainstem and spinal cord. Based on morphology and double-labeling experiments, white matter cells with TDP-43-positive inclusions most likely represent oligodendrocytes. Biochemically, hyperphosphorylated and truncated TDP-43 was detectable in insoluble brain extracts from affected white matter regions in FTLD-U, similar to the biochemical signature observed in FTLD-U gray matter. Taken together, these results expand the spectrum of TDP-43 pathology in FTLD-U, suggesting that white matter pathology might contribute to the neurodegenerative process and clinical symptoms in FTLD-U.

MeSH Terms
Adult Aged Antigens, CD/metabolism Antigens, Differentiation, Myelomonocytic/metabolism DNA-Binding Proteins/metabolism Dementia/metabolism,pathology Female Frontal Lobe/metabolism,pathology Glial Fibrillary Acidic Protein/metabolism Humans Immunohistochemistry Inclusion Bodies/metabolism Male Middle Aged Temporal Lobe/metabolism,pathology Ubiquitin/metabolism
Chemicals
Antigens, CD Antigens, Differentiation, Myelomonocytic CD68 antigen, human DNA-Binding Proteins Glial Fibrillary Acidic Protein Ubiquitin
Authors & Affiliations
11 authors, click to expand affiliations / ORCID
Neumann Manuela
Center for Neurodegenerative Disease Research, Department of Pathology and Laboratory Medicine, University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania, USA. Manuela.Neumann@med.uni-muenchen.de
Kwong Linda K
Truax Adam C
Vanmassenhove Ben
Kretzschmar Hans A
Van Deerlin Vivianna M
Clark Chrisopher M
Grossman Murray
Miller Bruce L
Trojanowski John Q
Lee Virginia M-Y
Article Info
Journal
Journal of neuropathology and experimental neurology
Abbr.
J Neuropathol Exp Neurol
ISSN
0022-3069
Published
2007-03-00
Pages
177-83
Language
English
Region
England
NLM ID
2985192R
Subset
IM
Grants
NIA NIH HHS · P01 AG017586 · United States
NIA NIH HHS · P01 AG019724 · United States
NIA NIH HHS · AG10124 · United States
NIA NIH HHS · AG17586 · United States
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