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PMID: 1701789 Published · ppublish English Journal Article Research Support, U.S. Gov't, P.H.S.

Aberrant expression of cytokine genes in peritoneal macrophages from mice infected with LP-BM5 MuLV, a murine model of AIDS.

Journal of immunology (Baltimore, Md. : 1950) ·Vol. 146 ·No. 1 ·1991-01-01 ·Pages 121-7

Cheung SC, Chattopadhyay SK, Hartley JW, Morse HC, Pitha PM

Abstract

Mice infected with LP-BM5 murine leukemia virus (MuLV) develop a syndrome denoted as murine AIDS. Macrophages harvested from the peritoneal cavities of these mice at 4 or 9 wk postinoculation with LP-BM5 MuLV were analyzed by Northern hybridization for the presence of the defective LP-BM5 virus and their ability to synthesize various cytokines upon induction with Newcastle disease virus (NDV) or (LPS). Neither IFN-alpha or IFN-beta was found to be constitutively expressed in LP-BM5-infected macrophages and in NDV induction studies, and the levels of biologically active IFN-alpha and its mRNA were found to be lower in LP-BM5 MuLV-infected macrophages than in the macrophages from uninfected controls. Similarly, after NDV or LPS induction, the levels of TNF mRNA and TNF protein were significantly lower in LP-BM5-infected macrophages than in macrophages from uninfected mice. The LP-BM5 MuLV-infected macrophages constitutively expressed low levels of IL-1 beta, and when induced with LPS, the relative levels of IL-1 beta were significantly higher in infected than in uninfected macrophages. Although no constitutive expression of IL-6 was detected, the levels of IL-6 mRNA induced with NDV were higher in LP-BM5 MuLV-infected macrophages than in controls. Thus, we found alterations in the expression of selected cytokines in macrophages from mice inoculated with LP-BM5 MuLV rather than a general deregulation of all cytokine expression. These results show that macrophages infected with the defective LP-BM5 virus respond differently to NDV- or LPS-stimulation and suggest that aberrant expression of certain cytokine genes may play a role in the immunopathologic condition in mice with murine AIDS.

MeSH Terms
Acquired Immunodeficiency Syndrome/genetics Animals Blotting, Northern Cytokines/genetics DNA-Binding Proteins/genetics Disease Models, Animal Gene Expression Interferon Regulatory Factor-1 Interferons/genetics Interleukins/genetics Leukemia Virus, Murine Macrophages/physiology Mice Mice, Inbred C57BL Peritoneal Cavity/cytology Phosphoproteins/genetics RNA, Messenger/genetics Transcription Factors/genetics Tumor Necrosis Factor-alpha/genetics
Chemicals
Cytokines DNA-Binding Proteins Interferon Regulatory Factor-1 Interleukins Irf1 protein, mouse Phosphoproteins RNA, Messenger Transcription Factors Tumor Necrosis Factor-alpha Interferons
Authors & Affiliations
5 authors, click to expand affiliations / ORCID
Cheung S C
Oncology Center, Johns Hopkins University School of Medicine, Baltimore, MD 21205.
Chattopadhyay S K
Hartley J W
Morse H C
Pitha P M
Article Info
Journal
Journal of immunology (Baltimore, Md. : 1950)
Abbr.
J Immunol
ISSN
0022-1767
Published
1991-01-01
Pages
121-7
Language
English
Region
United States
NLM ID
2985117R
Subset
IM
Grants
NCI NIH HHS · 5T32CA09243 · United States
NIAID NIH HHS · AI19737 · United States
NCI NIH HHS · CA50158 · United States
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