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PMID: 17003490 Published · ppublish English Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Pathological heterogeneity of frontotemporal lobar degeneration with ubiquitin-positive inclusions delineated by ubiquitin immunohistochemistry and novel monoclonal antibodies.

The American journal of pathology ·Vol. 169 ·No. 4 ·2006-10-00 ·Pages 1343-52

Sampathu DM, Neumann M, Kwong LK, Chou TT, Micsenyi M, Truax A, Bruce J, Grossman M, Trojanowski JQ, Lee VM

Abstract

Frontotemporal lobar degeneration with ubiquitin-positive inclusions (FTLD-U) is a common neuropathological subtype of frontotemporal dementia. Although this subtype of frontotemporal dementia is defined by the presence of ubiquitin-positive but tau- and alpha-synuclein-negative inclusions, it is unclear whether all cases of FTLD-U have the same underlying pathogenesis. Examination of tissue sections from FTLD-U brains stained with anti-ubiquitin antibodies revealed heterogeneity in the morphological characteristics of pathological inclusions among subsets of cases. Three types of FTLD-U were delineated based on morphology and distribution of ubiquitin-positive inclusions. To address the hypothesis that FTLD-U is pathologically heterogeneous, novel monoclonal antibodies (mAbs) were generated by immunization of mice with high molecular mass (Mr > 250 kd) insoluble material prepared by biochemical fractionation of FTLD-U brains. Novel mAbs were identified that immunolabeled all of the ubiquitin-positive inclusions in one subset of FTLD-U cases, whereas other mAbs stained the ubiquitin-positive inclusions in a second subset of cases. These novel mAbs did not stain inclusions in other neurodegenerative disorders, including tauopathies and alpha-synucleinopathies. Therefore, ubiquitin immunohistochemistry and the immunostaining properties of the novel mAbs generated here suggest that FTLD-U is pathologically heterogeneous. Identification of the disease proteins recognized by these mAbs will further advance understanding of molecular substrates of FTLD-U neurodegenerative pathways.

MeSH Terms
Aged Animals Antibodies, Monoclonal/biosynthesis,immunology Dementia/pathology Female Frontal Lobe/pathology Humans Immunohistochemistry Inclusion Bodies/chemistry Male Mice Middle Aged Temporal Lobe/pathology Ubiquitin/analysis,immunology
Chemicals
Antibodies, Monoclonal Ubiquitin
Authors & Affiliations
10 authors, click to expand affiliations / ORCID
Sampathu Deepak M
Center for Neurodegenerative Disease Research, Department of Pathology and Laboratory Medicine, University of Pennsylvania School of Medicine, 3600 Spruce St., 3rd Floor Maloney Bldg., Philadelphia, PA 19104, USA.
Neumann Manuela
Kwong Linda K
Chou Thomas T
Micsenyi Matthew
Truax Adam
Bruce Jennifer
Grossman Murray
Trojanowski John Q
Lee Virginia M-Y
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Article Info
Journal
The American journal of pathology
Abbr.
Am J Pathol
ISSN
0002-9440
Published
2006-10-00
Pages
1343-52
Language
English
Region
United States
NLM ID
0370502
PMCID
PMC1780184
Subset
IM
Grants
NIA NIH HHS · P30 AG010124 · United States
NIA NIH HHS · AG10124 · United States
NIA NIH HHS · P01 AG017586 · United States
NIA NIH HHS · T32 AG000255 · United States
NIA NIH HHS · AG17586 · United States
NIA NIH HHS · T32 AG 00255 · United States
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