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PMID: 15863510 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't

Ascochlorin inhibits matrix metalloproteinase-9 expression by suppressing activator protein-1-mediated gene expression through the ERK1/2 signaling pathway: inhibitory effects of ascochlorin on the invasion of renal carcinoma cells.

The Journal of biological chemistry ·Vol. 280 ·No. 26 ·2005-07-01 ·Pages 25202-9

Hong S, Park KK, Magae J, Ando K, Lee TS, Kwon TK, Kwak JY, Kim CH, Chang YC

Abstract

The expression of matrix metalloproteinases (MMPs) has been implicated in the invasion and metastasis of cancer cells. Here we examined the effect of ascochlorin, a prenyl-phenol anti-tumor compound from the fungus Ascochyta viciae, on the regulation of signaling pathways that control MMP-9 expression in human renal carcinoma (Caki-1) cells. Ascochlorin reduced the invasive activity of Caki-1 cells and inhibited phorbol 12-myristate 13-acetate-induced increases in MMP-9 expression and activity in a dose-dependent manner. Reporter gene, electrophoretic mobility shift, kinase inhibitor assays, and in vitro kinase assay showed that ascochlorin inhibits MMP-9 gene expression by suppressing activation of the nuclear transcription factor activator protein-1 (AP-1) via the extracellular signal-regulated kinase 1 and 2 pathway. The AP-1 family member most specifically affected by ascochlorin was Fra-1. Ascochlorin did not affect the activation of the c-Jun N-terminal or p38 kinase pathways. Moreover, transfection of Caki-1 cells with AP-1 decoy oligodeoxynucleotides resulted in the suppression of phorbol 12-myristate 13-acetate-induced MMP-9 expression and invasion. In conclusion, ascochlorin represents a unique natural anti-tumor compound that specifically inhibits MMP-9 activity through suppression of AP-1-dependent induction of MMP-9 gene expression.

MeSH Terms
Alkenes/pharmacology Amino Acid Motifs Binding, Competitive Blotting, Western Cell Line, Tumor Cell Nucleus/metabolism Cell Proliferation Dexamethasone/pharmacology Dose-Response Relationship, Drug Gene Expression Regulation, Enzymologic Genes, Reporter Humans JNK Mitogen-Activated Protein Kinases/metabolism Matrix Metalloproteinase 9/biosynthesis Mitogen-Activated Protein Kinase 1/metabolism Mitogen-Activated Protein Kinase 3/metabolism Models, Biological Models, Chemical Phenols/pharmacology Plasmids/metabolism Protein Structure, Tertiary Reverse Transcriptase Polymerase Chain Reaction Signal Transduction Tetradecanoylphorbol Acetate Thioctic Acid/pharmacology Transcription Factor AP-1/biosynthesis Transcription, Genetic Transfection p38 Mitogen-Activated Protein Kinases/metabolism
Chemicals
Alkenes Phenols Transcription Factor AP-1 Thioctic Acid Dexamethasone JNK Mitogen-Activated Protein Kinases Mitogen-Activated Protein Kinase 1 Mitogen-Activated Protein Kinase 3 p38 Mitogen-Activated Protein Kinases Matrix Metalloproteinase 9 Tetradecanoylphorbol Acetate ascochlorin
Authors & Affiliations
9 authors, click to expand affiliations / ORCID
Hong Sahyun
Department of Pathology and Department of Obstetrics and Gynecology, College of Medicine, Catholic University of Daegu, Daegu 705-034, Korea.
Park Kwan-Kyu
Magae Junji
Ando Kunio
Lee Tae-Sung
Kwon Taeg Kyu
Kwak Jong-Young
Kim Cheorl-Ho
Chang Young-Chae
Article Info
Journal
The Journal of biological chemistry
Abbr.
J Biol Chem
ISSN
0021-9258
Published
2005-07-01
Epub
2005-00-29
Pages
25202-9
Language
English
Region
United States
NLM ID
2985121R
Subset
IM
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