Pathologic effects of antimetabolites. I. Acute lesions in the hypothalamus, peripheral ganglia, and adrenal medulla caused by 3-acetyl pyridine and prevented by nicotinamide.

The American journal of pathology ·Vol. 31 ·No. 1 ·1955-00-00 ·Pages 189-99

HICKS SP

Abstract

暂无摘要

Keywords

ADRENAL MEDULLA/effect of drugs on HYPOTHALAMUS/pathology NERVES PERIPHERAL/pathology NICOTINIC ACID/derivatives PYRIDINES/toxicity

MeSH Terms

Adrenal Medulla/drug effects Antimetabolites Ganglia Hypothalamus/pathology Niacin/analogs & derivatives Niacinamide Nicotinic Acids Peripheral Nerves/pathology Pyridines/toxicity

Chemicals

Antimetabolites Nicotinic Acids Pyridines 3-acetylpyridine Niacinamide Niacin pyridine

Authors & Affiliations

1 authors, click to expand affiliations / ORCID

HICKS S P

References (2)

2 references, click to expand

Brain metabolism in vivo. II. The distribution of lesions caused by azide, malononitrile, plasmocid and dinitrophenol poisoning in rats.
AMA Arch Pathol. 1950 Nov;50(5):545-61 PMID: 14770718
Developmental brain metabolism; effects of cortisone, anoxia fluoroacetate, radiation, insulin, and other inhibitors on the embryo, newborn, and adult.
AMA Arch Pathol. 1953 Apr;55(4):302-27 PMID: 13030049

Article Info

Journal

The American journal of pathology

Abbr.

Am J Pathol

ISSN

0002-9440

Published

1955-00-00

Pages

189-99

Language

English

Region

United States

NLM ID

0370502

PMCID

PMC1942611

Subset

External Links

PubMed source PMC Full Text View journal details

Analysis Services

Pathologic effects of antimetabolites. I. Acute lesions in the hypothalamus, peripheral ganglia, and adrenal medulla caused by 3-acetyl pyridine and prevented by nicotinamide.

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