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PMID: 11586956 Published · ppublish English Case Reports Journal Article Research Support, Non-U.S. Gov't

Development of type 1 diabetes despite severe hereditary B-cell deficiency.

The New England journal of medicine ·Vol. 345 ·No. 14 ·2001-00-04 ·Pages 1036-40

Martin S, Wolf-Eichbaum D, Duinkerken G, Scherbaum WA, Kolb H, Noordzij JG, Roep BO

Abstract

暂无摘要

MeSH Terms

Adolescent Agammaglobulinaemia Tyrosine Kinase Agammaglobulinemia/complications,genetics Antigens, CD/analysis Autoantibodies/blood B-Lymphocytes/physiology Cell Division Cytokines/blood Diabetes Mellitus, Type 1/etiology,genetics,immunology Genetic Linkage Humans Male Protein-Tyrosine Kinases/genetics Sequence Analysis, DNA T-Lymphocytes/cytology,immunology X Chromosome

Chemicals

Antigens, CD Autoantibodies Cytokines Protein-Tyrosine Kinases Agammaglobulinaemia Tyrosine Kinase

Authors & Affiliations

7 authors, click to expand affiliations / ORCID

Martin S

German Diabetes Center, German Diabetes Research Institute, Heinrich Heine University, Düsseldorf. martin@dfi.uni-duesseldorf.de

Wolf-Eichbaum D

Duinkerken G

Scherbaum W A

Kolb H

Noordzij J G

Roep B O

Article Info

Journal

The New England journal of medicine

Abbr.

N Engl J Med

ISSN

0028-4793

DOI

10.1056/NEJMoa010465

martin@dfi.uni-duesseldorf.de

Published

2001-00-04

Pages

1036-40

Language

English

Region

United States

NLM ID

0255562

Subset

Corrections

CommentIn

PMID: 11586961

CommentIn

PMID: 11844863

CommentIn

PMID: 11858222

External Links

PubMed source DOI: 10.1056/NEJMoa010465 View journal details

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Development of type 1 diabetes despite severe hereditary B-cell deficiency.

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