Challenges of comparative expression profiling studies of complex diseases: mouse models of myocardial hypertrophy. Focus on "Divergent transcriptional responses to independent genetic causes of cardiac hypertrophy".

Physiological genomics ·Vol. 6 ·No. 1 ·2001-06-06 ·Pages 1-2

Glueck SB, Dzau VJ, Lee R, Pratt RE

Abstract

暂无摘要

MeSH Terms

Animals Cardiomegaly/genetics,metabolism Disease Models, Animal Gene Expression Profiling Mice Mice, Transgenic RNA, Messenger/biosynthesis Transcriptional Activation

Chemicals

RNA, Messenger

Authors & Affiliations

4 authors, click to expand affiliations / ORCID

Glueck S B

Dzau V J

Lee R

Pratt R E

Article Info

Journal

Physiological genomics

Abbr.

Physiol Genomics

ISSN

1531-2267

DOI

10.1152/physiolgenomics.2001.6.1.1

Published

2001-06-06

Epub

2001-00-06

Pages

1-2

Language

English

Region

United States

NLM ID

9815683

Subset

Corrections

CommentOn

PMID: 11395543

External Links

PubMed source DOI: 10.1152/physiolgenomics.2001.6.1.1 View journal details

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Challenges of comparative expression profiling studies of complex diseases: mouse models of myocardial hypertrophy. Focus on "Divergent transcriptional responses to independent genetic causes of cardiac hypertrophy".

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