Abstract
Matings of a number of Saccharomyces cerevisiae stocks give different output ratios of mitochondrial genotypes depending on whether the cells are glucose-repressed or derepressed. The effects of glucose repression are independent of cellular mating type and mitochondrial genotype, and take place at least in part after zygotes are formed. An explanation is proposed in terms of changes in the relative numbers of mitochondrial DNA molecules contributed by the a and alpha parents, modified by selective replication or destruction of molecules inside the zygote.
MeSH Terms
Chloramphenicol/pharmacology
Crosses, Genetic
Drug Resistance, Microbial
Erythromycin/pharmacology
Extrachromosomal Inheritance
Genes, Regulator
Genotype
Glucose/metabolism
Mitochondria
Mutation
Oligomycins/pharmacology
Recombination, Genetic
Saccharomyces cerevisiae/drug effects,metabolism
Chemicals
Oligomycins
Erythromycin
Chloramphenicol
Glucose
Authors & Affiliations
1 authors, click to expand affiliations / ORCID
Birky C W
References (9)
9 references, click to expand
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