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PMID: 10878289 Published · ppublish English Journal Article Review

Inhibition of p38 MAP kinase as a therapeutic strategy.

Immunopharmacology ·Vol. 47 ·No. 2-3 ·2000-05-00 ·Pages 185-201

Lee JC, Kumar S, Griswold DE, Underwood DC, Votta BJ, Adams JL

Abstract

Since the discovery of p38 MAP kinase in 1994, our understanding of its biology has progressed dramatically. The key advances include (1) identification of p38 MAP kinase homologs and protein kinases that act upstream and downstream from p38 MAP kinase, (2) identification of interesting and potentially important substrates, (3) elucidation of the role of p38 MAP kinase in cellular processes and (4) the establishment of the mechanism by which the pyridinylimidazole p38 MAP kinase inhibitors inhibit enzyme activity. It is now known that there are four members of the p38 MAP kinase family. They differ in their tissue distribution, regulation of kinase activation and subsequent phosphorylation of downstream substrates. They also differ in terms of their sensitivities toward the p38 MAP kinase inhibitors. The best-studied isoform is p38 alpha, whose activation has been observed in many hematopoietic and non-hematopoietic cell types upon treatment with appropriate stimuli. The pyridinylimidazole compounds, exemplified by SB 203580, were originally prepared as inflammatory cytokine synthesis inhibitors that subsequently were found to be selective inhibitors of p38 MAP kinase. SB 203580 inhibits the catalytic activity of p38 MAP kinase by competitive binding in the ATP pocket. X-ray crystallographic studies of the target enzyme complexed with inhibitor reinforce the observations made from site-directed mutagenesis studies, thereby providing a molecular basis for understanding the kinase selectivity of these inhibitors. The p38 MAP kinase inhibitors are efficacious in several disease models, including inflammation, arthritis and other joint diseases, septic shock, and myocardial injury. In all cases, p38 activation in key cell types correlated with disease initiation and progression. Treatment with p38 MAP kinase inhibitors attenuated both p38 activation and disease severity. Structurally diverse p38 MAP kinase inhibitors have been tested extensively in preclinical studies.

MeSH Terms
Animals Cytokines/drug effects,physiology Enzyme Inhibitors/pharmacology,therapeutic use Humans Imidazoles/pharmacology,therapeutic use MAP Kinase Signaling System/drug effects,physiology Mitogen-Activated Protein Kinases/antagonists & inhibitors,physiology Substrate Specificity Thiazoles/pharmacology,therapeutic use p38 Mitogen-Activated Protein Kinases
Chemicals
Cytokines Enzyme Inhibitors Imidazoles Thiazoles 6-(4-fluorophenyl)-2,3-dihydro-5-(4-pyridinyl)imidazo(2,1-b)thiazole Mitogen-Activated Protein Kinases p38 Mitogen-Activated Protein Kinases
Authors & Affiliations
6 authors, click to expand affiliations / ORCID
Lee J C
SmithKline Beecham Pharmaceuticals, 709 Swedeland Road, King of Prussia, PA 19406, USA. john_c_lee@sbphrd.com
Kumar S
Griswold D E
Underwood D C
Votta B J
Adams J L
Article Info
Journal
Immunopharmacology
Abbr.
Immunopharmacology
ISSN
0162-3109
Published
2000-05-00
Pages
185-201
Language
English
Region
Netherlands
NLM ID
7902474
Subset
IM
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