Abstract
We have examined the structure and fusion potential of the duck hepatitis B virus (DHBV) envelope proteins by treating subviral particles with deforming agents known to release envelope proteins of viruses from a metastable to a fusion-active state. Exposure of DHBV particles to low pH triggered a major structural change in the large envelope protein (L), resulting in exposure of trypsin sites within its S domain but without affecting the same region in the small surface protein (S) subunits. This conformational change was associated with increased hydrophobicity of the particle surface, most likely arising from surface exposure of the hydrophobic first transmembrane domain (TM1). In the hydrophobic conformation, DHBV particles were able to bind to liposomes and intact cells, while in their absence these particles aggregated, resulting in viral inactivation. These results suggests that some L molecules are in a spring-loaded metastable state which, when released, exposes a previously hidden hydrophobic domain, a transition potentially representing the fusion-active state of the envelope.
MeSH Terms
Animals
Binding Sites
Dithiothreitol/pharmacology
Ducks
Heating
Hepatitis B Virus, Duck/chemistry,drug effects,metabolism
Hydrogen-Ion Concentration
Liposomes/metabolism
Protein Conformation
Trypsin/metabolism
Viral Envelope Proteins/chemistry,metabolism
Virion/metabolism
Chemicals
L protein, hepatitis B virus
Liposomes
Pre-S protein, Duck hepatitis B virus
Viral Envelope Proteins
Trypsin
Dithiothreitol
Authors & Affiliations
2 authors, click to expand affiliations / ORCID
Grgacic E V
Macfarlane Burnet Centre for Medical Research and Australian Centre for Hepatitis Virology, Fairfield 3078, Victoria, Australia. grgacic@burnet.edu.au
Schaller H
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