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PMID: 10556104 Published · ppublish English Journal Article Research Support, U.S. Gov't, P.H.S.

Acid-induced lung injury. Protective effect of anti-interleukin-8 pretreatment on alveolar epithelial barrier function in rabbits.

American journal of respiratory and critical care medicine ·Vol. 160 ·No. 5 Pt 1 ·1999-11-00 ·Pages 1450-6

Modelska K, Pittet JF, Folkesson HG, Courtney Broaddus V, Matthay MA

Abstract

Although prior experimental work has demonstrated that anti-interleukin-8 (anti-IL-8) therapy reduces lung endothelial injury after acid instillation, there is no information regarding the effect of anti-IL-8 on the function of the alveolar epithelial barrier after acid-induced lung injury. Therefore, the primary objective of this study was to determine the effect of acid-induced lung injury on the function of the alveolar epithelium, and secondly to determine whether pretreatment with anti-IL-8 attenuates acid-induced injury to the lung epithelial barrier. Hydrochloric acid (pH = 1.5 in 1/3 normal saline) was instilled into the lungs of anesthetized, ventilated rabbits. Anti-IL-8 monoclonal antibody (2 mg/kg) or saline was given intravenously 5 min before acid instillation. Acid instillation into the distal airspaces caused an increase in the alveolar epithelial permeability to protein and an approximately 50% reduction in net alveolar fluid clearance. Because a decrease in net alveolar fluid clearance could be due to lung endothelial injury and increased fluid flux from the blood into the airspaces, additional experiments were carried out in which pulmonary blood flow was eliminated. In the absence of pulmonary blood flow, acid instillation led to a 50% decrease in net alveolar fluid clearance. Pretreatment with anti-IL-8 antibody significantly reduced the acid-mediated increase in bi-directional transport of protein across the alveolar epithelium and restored alveolar fluid clearance to normal. The results indicate that acid instillation causes injury to the alveolar epithelial barrier that can be distinguished from the injury to the lung endothelium. Furthermore, pretreatment with anti-IL-8 therapy prevents acid-induced alveolar epithelial injury, a finding of potential clinical importance.

MeSH Terms
Animals Antibodies, Monoclonal/administration & dosage Biological Transport, Active Capillary Permeability Extravascular Lung Water/physiology Hemodynamics Hydrochloric Acid Interleukin-8/immunology,physiology Lung/blood supply Male Permeability Pneumonia, Aspiration/complications,physiopathology Proteins/pharmacokinetics Pulmonary Alveoli/physiopathology Rabbits Recombinant Proteins Respiratory Distress Syndrome/etiology,physiopathology Respiratory Mucosa/physiopathology
Chemicals
Antibodies, Monoclonal Interleukin-8 Proteins Recombinant Proteins Hydrochloric Acid
Authors & Affiliations
5 authors, click to expand affiliations / ORCID
Modelska K
Department of Anesthesia, Cardiovascular Research Institute, University of California, San Francisco, California, USA.
Pittet J F
Folkesson H G
Courtney Broaddus V
Matthay M A
Article Info
Journal
American journal of respiratory and critical care medicine
Abbr.
Am J Respir Crit Care Med
ISSN
1073-449X
Published
1999-11-00
Pages
1450-6
Language
English
Region
United States
NLM ID
9421642
Subset
IM
Grants
NHLBI NIH HHS · HL 51854 · United States
NHLBI NIH HHS · HL-ES06331 · United States
Corrections
CommentIn
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