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题目:: Lack of chemokine signaling through CXCR5 causes mortality, ventricular dilatation and deranged matrix during pressure overload
ID:
状态:: 发布时间Oct. 1, 2010 , 更新时间 June 10, 2011
物种:: Mus musculus
摘要:: Inflammatory mechanisms have been suggested to play a role in the development of heart failure (HF), but a role for chemokines is largely unknown. The aim of this study was to analyze the role of the chemokine CXCL13 and its receptor CXCR5 in cardiac pathophysiology leading to HF A microarray study was performed on seven mice harboring a systemic knockout of the CXCR5 (CXCR5-/-) and seven wildtype mice. Four of the knockouts and four of the wild type mice were subjected to aorta banding and three of each to sham operations.
实验种类:: transcription profiling by array
样本量:: 14
实验设计:
: 无设计数据
数据号:: E-GEOD-22295, GSE22295
数据状态:: 无法自动分析，您可以尝试手动分析数据。

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